The 1982 treasure hunt book, The Secret, has clues to 12 hidden gems. Only two have been found. James Renner is on a quest to discover the others, and he invites you to join the hunt.Read the rest
There was no such epoch as “before the big bang,” because time began with the big bang, says physicist and astrobiologist Paul Davies.Read the rest
Someone is leaving coded messages and items such as a plastic leaf with paint dots inside envelopes between the pages of books at Western University. “It’s completely baffling!” says assistant professor Mike Moffatt, who has been tracking the messages on his blog.
The ocean is big and deep. The most likely scenario, right now, is that Malaysia Airlines Flight 370 crashed into the water and no one has yet looked in just the right place to find evidence of that crash. (You can read more about losing planes in the age of GPS in a post Rob made earlier today.) But the case made me curious about other lost planes — cases where an aircraft just "vanished" and nobody ever found a crash site or debris.
Naturally, Wikipedia has a list for that ...
Read the rest
The 600-year-old, strangely-illustrated Voynich Manuscript (which resides at Yale University) has been called the most mysterious manuscript in the world. Not a single word of the secret language has been decoded, at least not until now. Stephen Bax of the University of Bedfordshire says he has decoded ten words from the Voynich Manuscript. This seems to indicate that the document is not a hoax filled with nonsense words, as some scholars have concluded.
Stephen Bax, who teaches at the University of Bedfordshire, has produced a paper and a video where he details his theories on the text and provides translations of ten words from the manuscript, which are proper names of various plants that are depicted in the manuscript. Professor Bax explains, “I hit on the idea of identifying proper names in the text, following historic approaches which successfully deciphered Egyptian hieroglyphs and other mystery scripts, and I then used those names to work out part of the script.
I have not yet watch Bax's 47-minute video, above.
I don't remember how I found out about Munseys. It's a website with links to thousands of out-of-print books, with over 1,500 pulp era novels. There's a lot of good stuff to be found here, as well as much that fits somewhere between excellent and awful. I downloaded and read a so-so novel called Alley Girl, by Jonathan Craig. There's no "alley girl" in it, which I didn't care about anyway. It's a about a hard-boiled sociopathic cop who tries to set up an innocent man for the electric chair so he can profit in more ways that one. It took me about an hour and a half to read and it made me forget about my 18-hour plane ride last week.
At the top of the chart, get Charles Willeford's Whip Hand (he ghost wrote it for Frank Sanders, or at least wrote most of it). It's about a kidnapping/murder seen through the eyes of different characters, and takes places in Texas. Willeford is one of the great hardboiled noir writers. I read everything of his I can get my hands on.
Like Gone Girl (a novel I liked so much that I interviewed the author, Gillian Flynn, on Gweek), The Silent Wife is told in alternating chapters from the points-of-view of a common-law husband and wife whose union has endured two decades of infidelity and stifled communication.
Jodi is a 42-year-old part-time psychologist who has developed a coping mechanism for her husband Todd's philandering: she gaslights him with little annoyances. For instance, she removes a key from his keyring so he can't enter his office building. But other than the quiet tricks she plays on Todd, she seems to enjoy his company and delights in making gourmet meals that they both enjoy in their high-end Chicago riverfront condominium.
Todd is a fairly well-to-do property developer, and incorrigible pleasure-seeker. He's a charming dinner party host and everyone likes him, in part because he avoids conflict at all costs. He knows that Jodi knows about his frequent dalliances, but neither he nor Jodi ever bring it up in conversation.
Read the rest
Read the rest
Besides magnetism, there's another thing that the Insane Clown Posse was on-track in categorizing as a mind-blowing mystery — Why do Shaggy 2 Dope's kids look just like him? As with the magnets, this is another situation where the obvious answer (it's genetics!) masks a much more complicated issue that science hasn't totally figured out yet. At Pacific Standard, Michael White explains why genetics is still messing with our heads, almost 150 years after Mendel:
The problem: most of the genetic differences discovered have only a very small effect. And when you add up all those effects, the result can’t possibly explain the full influence of our genes on those traits. For example, researchers have identified hundreds of DNA differences between people that influence the very strongly heritable trait of human height, but the total effect of those differences added together explains only about 10 percent of the genetic influence on height. In other words, we still can’t explain why tall parents have tall children.
Scientists have named this discrepancy the “missing heritability,” and they’ve spent the last half-decade trying to find it.
The woman pictured above (on the left in the early 2000s, on the right in a 1990 passport photo) committed suicide in 2010. When she died, she took with her the knowledge of who she actually was — the private detective hired by her ex-husband's family is now calling her Jane Doe.
When she married, in 2004, she was known as Lori Erica Kennedy. After her death, her ex-husband discovered a lock box containing documents that showed she had changed her name to that from Becky Sue Turner. But here's the twist. She wasn't actually Becky Sue Turner, either. The real Becky Sue Turner had died in a house fire in 1971, at age 2, and her family had never seen this woman before.
So who is Jane Doe? That's what her ex-husband and his family are trying to figure out. When she died, she left behind a young daughter, who is going to want answers someday. Meanwhile, the mystery is seriously curiosity provoking. As far as anyone can tell, her trail goes dead in 1988. Her entire life before then is a great big blank.
The Seattle Times has the full story, including copies of documents and photos found in the lockbox and a timeline of what we do know about Jane Doe.
The Geminids are one of the big deal meteor showers that happen every year. In fact, they're regarded as one of the most reliable and impressive. They're also a little strange.
Most meteor showers happen when Earth and a comet cross paths, slingling rocks, dust, and debris from the comet's tail into our atmosphere. The sudden influx of shooting starts that results is a highly noticeable event and humans have been recording them for millennia.
The Geminids are different. They sort of just appeared, seemingly out of nowhere, back in 1862. And it wasn't until the 1980s that scientists were finally able to identify the thing that was producing them. At which point, ish got weirder.
That's because the object, known as 3200 Phaethon, is really confusing. It doesn't seem to be a comet. At least, not a normal, healthy, functioning comet. It doesn't even have a tail. In fact, at this point most scientists think it's probably an asteroid, which then leads to still-yet-unexplained question of where all the meteors come from. Asteroids, after all, do not typically accumulate tails of small rocks. So far, the best guess has to do with 3200 Phaethon's orbit, which over the course of about a year and a half takes it closer to the Sun than Mercury and then back out further from the Sun than Mars. Those wild temperature swings might lead to the asteroid cracking and throwing off dust and debris, which then becomes meteors. But, as a NASA info page pointed out in 2010, that explanation doesn't totally cut it.
The amount of dust 3200 Phaethon ejected during its 2009 sun-encounter added a mere 0.01% to the mass of the Geminid debris stream—not nearly enough to keep the stream replenished over time.
According to the International Meteor Organization, you can expect the Geminids to peak tomorrow night, around 5:30 pm, Central Time. But this is a big shower, so you're likely to see something even if you can't hit the exact peak.
Also: While you're watching for meteors, also keep an eye out for an upcoming feature here by Miles O'Brien, which will delve into the latest in Geminid science!
Make use of The Bad Astronomy guide to meteor watching
The Curiosity rover comes complete with a mini chemistry lab. It's designed to analyze the composition of Martian soils and Martian air. And, right now, that particular piece of equipment is at the center of a giddy storm of activity. Curiosity has turned up something important — big enough for Curiosity's principal investigator to tell NPR, "This data is gonna be one for the history books."
What is it? NASA's not telling just yet. Right now, researchers are in the process of verifying said exciting data, in order to make sure they aren't deceiving themselves into thinking they've spotted something that isn't really there. That's pretty good policy, given the recent flap around over-hyped studies about Earth-like planets and arsenic-based life.
On the other hand, if you're trying to avoid overhyping something, might I suggest that "We have groundbreaking, world-changing data that we can't tell you about yet," is maybe not the best way to do it.
Pictured: A 360-degree view of Mars, taken by Curiosity on October 5th, from the location where it first started collecting samples of rocks and dirt. NASA/JPL
Over the past few years, multiple people have died in Thailand from what appears to be exposure to some kind of poison. Most of these people have been tourists. And most of them have been young women. The deaths have happened in clusters. Five or so on the island vacation hotspot of Kho Phi Phi. Another group of six at Chiang Mai's Downtown Inn.
Lots of possible explanations have been suggested — ranging from serial killers, to hallucinogenic beach drinks, to overuse of banned insecticides in hotel rooms. But, so far, none of the specific poisons proposed as the culprit totally makes sense in relation to the deaths. And, to make things worse, it seems like Thai authorities are doing their best to make it difficult to actually investigate what has happened in individual cases, and figure out whether the cases are linked or not. At this point, it's hard to even know whether all the people who have died exhibited the same symptoms.
Deborah Blum, a Pulitzer-winning journalist who has done a lot of reporting on poisons and true crime has been following this story and just published another piece on the still-unfolding mess.
Your daughter died.
Your daughter died thousands of miles from home. In a hotel where no one came to help. In a hospital where she struggled to keep breathing and just couldn’t. In a room where her heart – and somehow you still don’t really believe this – just stuttered to a stop. In a country, where authorities have failed for months, years even, to tell you how or why your daughter died.
Your daughter, you’ve come to realize, died in a pattern that links too many other young women, a chain of suspected poisonings over the last few years. Jill St. Onge, 27, of Seattle, Washington, and Julie Bergheim, 22, of Drammen, Norway, who both died in May 2009 on the southern island of Koh Phi Phi. Sherifa Khalid, 24, of Kuwait, who died 12 hours she spent a day on the same island in July of the same year.
Rabies isn't funny. But, somehow, Heather Swain makes her story about her brush with rabies absolutely hilarious.
Now Swain didn't have rabies, but she did have a lot of the symptoms that go along with it. In this Story Collider piece, she talks about her experience being "that patient with a mystery disease" and what it's like when nobody actually figures out what's making you sick.
See those weird, black, spidery things dotting the dunes in this colorized photo taken by the Mars Reconnaissance Orbiter in 2010? Yeah. Nobody knows what the hell those things are.
What we do know about them just underlines how incredibly unfamiliar Mars really is to us. First spotted by humans in 1998, these splotches pop up every Martian spring, and disappear in winter. Usually, they appear in the same places as the previous year, and they tend to congregate on the sunny sides of sand dunes — all but shunning flat ground. There's nothing on Earth that looks like this that we can compare them to. It's a for real-real mystery, writes Robert Krulwich at NPR. But there are theories:
Scientists from the U.S. Geological Survey, from Hungary, from the European Space Agency have all proposed explanations; the leading one is so weird, it's transformed my idea of what it's like to be on Mars. For 20 years, I've thought the planet to be magnificently desolate, a dead zone, painted rouge. But imagine this: Every spring, the sun beats down on a southern region of Mars, morning light melts the surface, warms up the ground below, and a thin, underground layer of frozen CO2 turns suddenly into a roaring gas, expands, and carrying rock and ice, rushes up through breaks in the rock, exploding into the Martian air. Geysers shoot up in odd places. It feels random, like being surprise attacked by an monstrous, underground fountain.
"If you were there," says Phil Christensen of Arizona State University, "you'd be standing on a slab of carbon dioxide ice. All around you, roaring jets of carbon dioxide gas are throwing sand and dust a couple hundred feet into the air." The ground below would be rumbling. You'd feel it in your spaceboots.
Read the rest of Robert Krulwich's post — and check out some spectacular photos of the things — at NPR
Marine biologists think they can identify this strange, blobby creature that floated up out of the ocean depths and was captured on an oil drill video. That, alone, is not terribly surprising. Marine biologists know a lot of weird ocean creatures.
But would you believe they identified it by its gonads?
When I was little, I read a Reader's Digest book of great disasters, which included a segment on the Black Death. One of the things the book tried to do was explain, on a child's level, why it wasn't easy to figure out that rats and fleas were the source of the plague. You couldn't just look for patterns, because there seemed to be no pattern. Half a household might drop dead while the other half only got a little sick, or remained entirely healthy. Plague doctors who handled the sick every day lived another 20 years. The real spread of disease wasn't like the movies, where one person coughing means everyone in close proximity is doomed.
One reason for the emergence of strange non-patterns like this is something called "super spreaders"—basically, some people spread disease more effectively than others. The infamous Typhoid Mary is the poster child for super spreaders, but the effect has been well-documented in a range of infectious diseases and it goes beyond the simple story of one woman who infected thousands. In fact, what makes the super spreader phenomenon so fascinating is that it isn't an anomaly at all. Super spreaders are the primary way some diseases spread. The Contagions blog—which is all about the history of infectious disease—has a great post up about this.
Eventually new models arose like the “20/80″ rule that says that 20% of cases are responsible for 80% of the transmission and formed a core ‘high risk’ group. This model works well for some diseases but not all.
For pathogens that do rely on super-spreaders, the majority of cases will not transmit the infection to anyone. This can lead to a sense of false security because it seems poorly communicated. As Galvani and May assert, “heterogeneously infectious emerging disease will be less likely to generate an epidemic, but if sustained, the resulting epidemic is more likely to be explosive”. Super-spreaders tend to beget more super-spreaders, although most of the cases they generate will still not transmit the infection to anyone. For example, a super-spreader begets 30 cases, 3 (10%) of which become new super spreaders. The rest may transmit to 0-1 people.
Super-spreading has been documented for HIV, SARS (Sudden Acute Respiratory Syndrome), measles, malaria, smallpox and monkeypox, pneumonic plague, tuberculosis, Staphylococcus aureus, typhoid fever, and a variety bacterial sexually transmitted diseases.
And that brings us back to medical mysteries because, the Contagion blog explains, we don't know exactly why some people are super spreaders and others aren't—or why some people are more vulnerable to infection than others. So far, what we have to go on is a list of well-established correlations.
I recently had what I am pretty sure was foodborne illness. It arrived in the middle of a friend's birthday party, a sudden onslaught of misery that lasted for the next 8 hours, reminding me, horribly, of a similar scene in The Mask of the Red Death. It was followed by two days of pretty much constant sleep. I don't recommend it.
But if a growing body of research is right, that 48-hours of grossness might not be the end of your body's interaction with a foodborne bug. In fact, some people seem to have otherwise unexplained symptoms persisting for years after they thought they'd recovered from food poisoning. This is best documented in people whose food poisoning experience was much worse than mine—folks who ended up in the hospital or the doctor's office and were, thus, accurately diagnosed, so we know they had a foodborne illness and not, say, a stomach flu. But it's an interesting hypothesis.
Maryn McKenna, my favorite Scary Disease Girl, has a story about this at Scientific American, plus some extra information at her Wired blog, where she explains why this phenomenon is so difficult to study.
I start the story with the tale of a Florida teen named Dana Dziadul, who 11 years ago was hospitalized with Salmonella and now at 14 has what is called “reactive” arthritis. Her mother Colette struggled for years to figure out why this was happening to her daughter, but didn’t put the pieces together until she was asked to complete a survey of foodborne illness survivors, and spotted a list of possible after-effects — sequelae, technically — that the surveyors were curious about. That caused her to go back into Dana’s medical chart, where she realized that her daughter’s joint problems actually began while she was hospitalized as a 3-year-old.
The challenge of proving this connection is that our system for investigating foodborne illness is not set up for tracking victims long afterward. That’s first because state health departments, which bear the burden of identifying outbreaks, are most concerned with finding people at the time, not keeping track of them; and second, because many outbreaks are spread across multiple states, with only a few victims in each state — so that maintaining contact with former victims would require a shared effort that no one is set up, or funded, to do. (That’s not even to mention the complication of people moving from one jurisdiction to another. Myself, for instance, I’ve moved five times in the past 10 years.)
This is an awkward sort of "How To" post because nobody really knows the answer. Here's the rather bleak reality: Rabies is not, typically, something you live through. If you think you've been exposed, you can get a life-saving vaccine. But, if you miss that window, and symptoms start to appear, your chances of survival are pretty much nil.
There are exceptions. We've talked before here about the Milwaukee Protocol, a medical intervention that some doctors think has allowed a handful of people to escape death. But the Milwaukee Protocol is not a simple thing. It involves hospitalization, as doctors put the affected person into a coma. Basically, they reboot the system. And it might not be as effective as we think it is. Five people have gotten the Milwaukee Protocol and survived. Thirty-two others received the treatment and still died. There's a lot of effort that goes into the Milwaukee Protocol, and it might be wasted effort.
Then there's this: In 2009, a teenage runaway walked into a Texas hospital, exhibiting symptoms of rabies—a diagnosis that was later confirmed. She didn't get vaccinated before symptoms started. She never got the Milwaukee Protocol. Instead, she recovered on her own and left the hospital hale and healthy three weeks later.
That's not supposed to happen in humans.
The Austin American-Statesman has a fascinating story about this case. At the heart is a big mystery. The young woman cut off contact with hospital researchers shortly after she was discharged from the hospital. She was homeless—in fact, she'd probably contracted rabies from a bat while camping in a cave. There's a very good chance that we may never know why she survived when so many others don't.
Veterinarians know that not all animals die after exposure — usually from a bite — to the rabies virus, which is carried in saliva and other bodily fluids. Much depends on how much of the virus is transmitted, the severity and location of the bite, and the immune system of the bitten animal.
"Abortive rabies is not unusual in research animals — it happens spontaneously all the time," Rupprecht said.
Rupprecht said the blood of certain human populations — maybe 1 percent of raccoon hunters, for example — also tests positive for rabies antibodies, indicating they have been exposed to the virus without becoming acutely ill. In the Amazon, where indigenous tribes are at daily risk of rabies from vampire bats, "it could be in the 10 percent or more range," he said.
The rabies vaccine, injected in a series of five doses, prevents onset of the disease if given soon enough after exposure. About 35,000 people receive the expensive shots — the series costs about $1,600 — in the U.S. each year. Seldom do more than a handful of people die from rabies in a year. But once rabies symptoms appear, the vaccine is no help.
"We don't have poster children. In our field, 99.9 percent of our cases are in the ground," Rupprecht said.
This is the town of Kivalina, Alaska. Last fall, when the ocean water that almost surrounds the town started turning a gooey orange, people (understandably) got a bit freaked out.
After ruling out the scarier options—i.e.,chemical pollution and toxic algae—scientists eventually pinned the orange tide on the presence of a plant fungus. And they turned up some good news: The fungus wasn't dangerous to people or ocean life.
Now, months later, researchers have identified what, exactly, the fungus is and where it was coming from. There's a fascinating detective story here, because, as Jennifer Frazer points out on Scientific American's Artful Amoeba blog, it's rather surprising that there was a fungal epidemic big enough to turn a whole port orange and nobody noticed it on the plants.
[But] Perhaps someone did.
Last October, David Wartinbee, a professor of aquatic biology at Kenai Peninsula College in Alaska’s south-central Kenai Peninsula, emailed me to say he’d seen something strange, and wondered if it might be the same thing that hit Kivalina. Though his neck of the woods is over 600 miles southeast from Kivalina as the snow goose flies, it’s not inconceivable they could be one in the same in a place so far north.
In early September, Wartinbee traveled 70 miles west to a place called the Twin Lakes by float plane (reputedly the SUV of Alaska). He saw an orange film on the water, and the spruce needles on nearby trees were clearly poxed with something.
You can read the rest of this story (and see Wartinbee's photos!) at The Artful Amoeba.
“If it exists, it has to be there. And if it’s not there, it will be known to be science fiction by December." — Vivek Sharma, a physics professor at UC San Diego, talking about the as-yet elusive Higgs boson particle. The search for the Higgs boson has recently been narrowed down. Basically, we've gone from looking for a needle in a haystack, to looking for a needle inside a small, hay-stuffed pillow.